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Herpesviruses 1 and 2 affect primarily the oral and genital areas, respectively Seroprevalence to both viruses increases with age; seroprevalence to HSV-2 increases with sexual activity Disease is typically a manifestation of reactivation, and the triggers for clinical reactivation are not completely understood Although HSV-1 is increasingly recognized as causing primary urogenital infections (> 65 70% in series from Israel and the United States), genital recurrences are much more frequent with HSV-2 Following the first year after infection, HSV-1 recurrences are rare, while HSV-2 reactivations gradually decrease Risk factors for HSV transmission include black race, female gender, a history of sexually transmitted infections (STIs), an increased number of partners, contact with commercial sex workers, lower socioeconomic status, young age of onset of sexual activity, and total duration of sexual activity Asymptomatic shedding of either virus is common, especially following primary infection or symptomatic recurrences, and may be responsible for transmission HSV-2 increases the risk of HIV acquisition, and in advanced HIV infection, HSV reactivates more frequently

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You should restructure your code so that it can tell you exactly what's wrong with the upload . Something like this: protected void ...

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HSV-1 mucocutaneous disease largely involves the mouth and oral cavity ( herpes labialis or gingivostomatitis; the latter largely in children) HSV-2 is the most common cause of genital ulcers in the developing world Digital lesions (whitlows) are an occupational hazard in medicine, nursing, and dentistry Vesicles form moist ulcers after several days and epithelialize over 1 2 weeks if left untreated Primary infection is usually more severe than recurrences but may be asymptomatic Recurrences often involve fewer lesions, tend to be labial, heal faster, and are induced by stress, fever, infection, sunlight, chemotherapy (eg, fludarabine), or other undetermined factors HSV-2 lesions largely involve the genital tract Virus remains latent in presacral ganglia Typical lesions are multiple, painful, small, grouped, and vesicular A manifestation of primary infection, predominantly in women, may be aseptic

1 Herpesviruses 1 & 2

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Spectrum of illness from stomatitis and urogenital lesions to facial nerve paralysis (Bell s palsy) and encephalitis Variable intervals between exposure and clinical disease, since HSV causes both primary (which may be subclinical) and reactivation disease Successful management with acyclovir or related acyclic compounds (valacyclovir, famciclovir)

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meningitis Asymptomatic HSV-2 occurs frequently in the CNS Urinary retention and chronic neuropathic pelvic pain may occur Proctitis and sacral lesions are particular problems in HIV-infected patients and may present with extensive, ulcerating, weeping lesions in patients with depressed CD4 counts Large ulcerations and atypical lesions suggest drug-resistant isolates (see below) Diagnosis is usually made clinically, but viral cultures of vesicular fluid or direct fluorescent antibody staining of scraped lesions remain the standard of diagnosis HSV can be identified using polymerase chain reaction (PCR), and its presence correlates with clinical reactivation The presence of intranuclear inclusion bodies and multinucleated giant cells on a Tzanck preparation or Calcofluor stain is supportive of a diagnosis of herpetic viral infection In STI, perinatal, and HIV clinics, type-specific serologies are useful for counseling HIV-infected individuals; pregnant women; sexual partners of persons infected with HSV; and those with an uncertain clinical history, high-risk behavior, or increased number of sexual partners

vors suffering neurologic sequelae HSV-1 predominantly causes herpes simplex encephalitis while HSV-1 and HSV-2 are both associated with benign recurrent lymphocytic (Mollaret s) meningitis

Disseminated HSV infection occurs in the setting of immunosuppression, either primary or iatrogenic, including corticosteroid usage, or rarely with pregnancy Skin lesions are a particular complication in patients with atopic eczema (eczema herpeticum) In disseminated disease, skin lesions are not always present

public static void Main( ) { for (int counter = 1; counter <= 100; counter++) { Console.Write("{0} ", counter); if (counter % 10 == 0) { Console.WriteLine("\t{0}", counter); } } } } }

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Esophagitis from HSV-1 in AIDS and other immunocompromised patients is diagnosed by endoscopic biopsy and cultures CMV esophagitis is distinguished by the size and depth of the lesion (smaller and deeper for HSV) HSV-1 is also postulated to activate mononuclear cells in the pathogenesis of achalasia

HSV can cause keratitis, blepharitis, and keratoconjunctivitis Keratitis is usually unilateral, is suggested by impaired visual acuity, and is diagnosed by branching (dendritic) ulcers that stain with fluorescein It may be difficult to differentiate HSV conjunctivitis clinically from adenoviral conjunctivitis in the acute stage Recurrences are frequent

Herpes simplex viruses remain, with drugs, the leading association with erythema multiforme and with the more severe, mucosally involved Stevens Johnson syndrome

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Both herpesviruses can infect the fetus and induce congenital malformations (organomegaly, bleeding, and CNS abnormalities) Neonatal transmission during delivery is more common than intrauterine infection Maternal infection during the third trimester is associated with the highest risk of neonatal transmission; about 70% of these infections are asymptomatic or unrecognized Invasive fetal monitoring and vacuum or forceps delivery can facilitate the risk of transmitting herpes

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