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received information. Route reports are processed only by those sent by a known neighbor; otherwise, they are discarded. Generally, two rules are followed: If the route entry is new and the metric is less than infinity, the route is added. That s the simple one. The second rule is tougher and is shown in the table. If the route entry exists, perform the following checks: If New Metric < infinity AND New metric > existing metric If New Metric < infinity AND New metric < existing metric If New Metric < infinity AND New metric = existing metric If New Metric = infinity AND New gateway = existing gateway If New Metric = infinity AND New gateway not equal to existing gateway If New metric is between route infinity and 2x infinity If the same neighbor is reporting it, update the entry; otherwise, discard the entry. Update the entry with the route and if necessary, update the reporting neighbor. Refresh the route and if the new neighbor has a lower IP address, update that entry. Route is now unreachable, update the entry. Ignore

reason for medical skepticism concerning CFS is that many studies examining cellular immune response abnormalities in people with CFS have not been consistently reproduced (Krupp, Mendelson, & Friedman, 1991) In reality, rarely is there a perfect biological test for an illness With CFS, there does appear to be mounting evidence of brain and immune system abnormalities (Komaroff, 2000a) It is possible that low levels of circulating cortisol suggest a condition of mild adrenal insuf ciency In individuals with CFS, this condition could stimulate immune activation, which could contribute to brain dysfunction (Komaroff, 2000b) Although no virus has been identi ed as the cause of CFS, the immune pattern seems to be ghting a virus, as evidenced by the RNase-L pathway.

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The third factor, Inhibition, was defined by intimacy problems and restricted expression of inner experiences and feelings The factor correlated negatively with NEO-PI-R and EPQ extraversion and resembled the DSM-IV avoidant and schizoid personality disorders The fourth factor, Compulsivity, clearly resembled NEO-PI-R conscientiousness and DSM-IV obsessivecompulsive personality disorder Congruency coefficients computed between the genetic and phenotypic factors on Emotional Dysregulation, Dissocial, Inhibition, and Compulsivity were 97, 97, 98, and 95, respectively The congruence between factors extracted from the phenotypic and nonshared environmental matrices were also high: 99, 96, 99, and 96, respectively This method was also applied to domains of the five-factor model assessed by Costa and McCrae s (1992) NEO-PI-R scale Jang, Livesley, Angleitner, Riemann, and Vernon (2002) estimated the genetic and environmental correlations between all 30 of the NEOPI-R facet scales in two independent samples of twins recruited in Germany and Canada.

Increasing evidence also points to neurological ndings including hyperintense signals on MRI scans, reductions in cerebral blood ow on SPECT scans (Lange, Wang, DeLuca, & Natelson, 1998), and autonomic dysfunction (primarily orthostatic intolerance and neurally mediated hypotension; Schondorf & Freeman, 1999) The onset of CFS is often linked with the recent presence of an infection CFS has been reported following acute mononucleosis (a viral infection), Lyme disease (a bacterial infection), and Q fever (an infection with a different type of infectious agent; Komaroff, 2000b) Activation by macrophages due to a virus or bacteria produces a release of interleukin 1, which causes an alteration in the electrical activity of the brain; it also causes a number of behavioral changes (eg.

Factor analysis of these matrices yielded five factors that were clearly recognizable as Neuroticism, Extraversion, Openness to Experience, Agreeableness, and Conscientiousness The correlation between the five genetic factors and the five normative factor structure was high at 83, 72, 92, 88, and 70 This correspondence between the genetic and observed factor structure of the NEO-PI-R suggests that all of the constituent parts of each broad domain share a common genetic basis These findings clearly indicate that the phenotypic structure of personality closely resembles the underlying genetic architecture, and that this observation is consistent across different measures This congruence also applies to personality disorder an observation that is especially interesting given the general lack of correspondence between genotype and phenotype for most mental disorders (Merikangas, 2002) It appears that the genetics.

, decreases in activity and social interaction, somnolence) designed to reduce unnecessary energy expenditure, so that available energy stores can be used to ght the infection (Maier, Watkins, & Fleshner, 1994) These cytokines might induce a state of chronic activation, which leads to a depletion of the stress hormone axis, and to other neuropsychiatric and neuroendocrine features associated with CFS (Saphier, 1994) Some individuals might be at higher risk of developing this chronic activation due to genetic vulnerabilities, or to constitutional or psychological factors Individuals with CFS might have hyperactive premorbid lifestyles, and this high action-proneness might be a predisposing factor for developing CFS (Van Houdenhove, Onghena, Neerinckx, & Hellin, 1995) Individuals who are chronically stressed have a persistent lack of cortisol, and this might also contribute to CFS (Heim, Ehlert, & Hellhammer, 2000).

In addition, stress might be a conditioned stimulus that leads to an impaired immune response (Cohen, Moynihan, & Ader, 1994) In addition, environmental factors might play a role in the etiology of CFS: People with CFS have signi cantly higher levels of chlorinated hydrocarbons than do control subjects (Dunstan et al, 1995)..

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